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Showing 1 to 12 of 820 entries
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ApoE deficiency promotes colon inflammation and enhances inflammatory potential oxidized-LDL and TNF-α in colon epithelial cells.

Bioscience reports

El-Bahrawy A, Tarhuni A, Kim H, Subramaniam V, Benslimane I, Abd Elmageed ZY, Okpechi S, Ghonim M, Hemeida R, Abo-Yousef A, El-Sherbiny G, Abdel-Raheem I, Kim J, Naura AS, Boulares H.
PMID: 27538678
Biosci Rep. 2016 Oct;36(5). doi: 10.1042/BSR20160195. Epub 2016 Aug 18.

Although deficiency in Apolipoprotein E (ApoE) is linked to many diseases, its effect on colon homeostasis remains unknown.  ApoE appears to control inflammation by regulating NF-kB.  This study was designed to examine whether ApoE deficiency affects factors of colon...

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El-Bahrawy A, Tarhuni A, Kim H, et al. ApoE deficiency promotes colon inflammation and enhances inflammatory potential oxidized-LDL and TNF-α in colon epithelial cells. Biosci Rep. 2016;36(5)doi: 10.1042/BSR20160195.
El-Bahrawy, A., Tarhuni, A., Kim, H., Subramaniam, V., Benslimane, I., Abd Elmageed, Z. Y., Okpechi, S., Ghonim, M., Hemeida, R., Abo-Yousef, A., El-Sherbiny, G., Abdel-Raheem, I., Kim, J., Naura, A. S., & Boulares, H. (2016). ApoE deficiency promotes colon inflammation and enhances inflammatory potential oxidized-LDL and TNF-α in colon epithelial cells. Bioscience reports, 36(5), . https://doi.org/10.1042/BSR20160195
El-Bahrawy, Ali, et al. "ApoE deficiency promotes colon inflammation and enhances inflammatory potential oxidized-LDL and TNF-α in colon epithelial cells." Bioscience reports vol. 36,5 (2016). doi: https://doi.org/10.1042/BSR20160195
El-Bahrawy A, Tarhuni A, Kim H, Subramaniam V, Benslimane I, Abd Elmageed ZY, Okpechi S, Ghonim M, Hemeida R, Abo-Yousef A, El-Sherbiny G, Abdel-Raheem I, Kim J, Naura AS, Boulares H. ApoE deficiency promotes colon inflammation and enhances inflammatory potential oxidized-LDL and TNF-α in colon epithelial cells. Biosci Rep. 2016 Oct;36(5). doi: 10.1042/BSR20160195. Epub 2016 Aug 18. PMID: 27538678; PMCID: PMC5052706.
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Defining the extreme substrate specificity of .

Bioscience reports

Bansal S, Durrett TP.
PMID: 27688773
Biosci Rep. 2016 Nov 08;36(6). doi: 10.1042/BSR20160277. Print 2016 Dec.

No abstract available.

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Bansal S, Durrett TP. Defining the extreme substrate specificity of . Biosci Rep. 2016;36(6)doi: 10.1042/BSR20160277.
Bansal, S., & Durrett, T. P. (2016). Defining the extreme substrate specificity of . Bioscience reports, 36(6), . https://doi.org/10.1042/BSR20160277
Bansal, Sunil, and Durrett, Timothy P. "Defining the extreme substrate specificity of ." Bioscience reports vol. 36,6 (2016). doi: https://doi.org/10.1042/BSR20160277
Bansal S, Durrett TP. Defining the extreme substrate specificity of . Biosci Rep. 2016 Nov 08;36(6). doi: 10.1042/BSR20160277. Print 2016 Dec. PMID: 27688773; PMCID: PMC5100001.
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Correction: Fangjing decoction relieves febrile seizure-induced hippocampal neuron apoptosis in rats via regulating the Akt/mTOR pathway.

Bioscience reports

[No authors listed]
PMID: 30670672
Biosci Rep. 2019 Jan 22;39(1). doi: 10.1042/BSR-20181206_COR. Print 2019 Jan 31.

No abstract available.

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Correction: Fangjing decoction relieves febrile seizure-induced hippocampal neuron apoptosis in rats via regulating the Akt/mTOR pathway. Biosci Rep. 2019;39(1)doi: 10.1042/BSR-20181206_COR.
(2019). Correction: Fangjing decoction relieves febrile seizure-induced hippocampal neuron apoptosis in rats via regulating the Akt/mTOR pathway. Bioscience reports, 39(1), . https://doi.org/10.1042/BSR-20181206_COR
"Correction: Fangjing decoction relieves febrile seizure-induced hippocampal neuron apoptosis in rats via regulating the Akt/mTOR pathway." Bioscience reports vol. 39,1 (2019). doi: https://doi.org/10.1042/BSR-20181206_COR
Correction: Fangjing decoction relieves febrile seizure-induced hippocampal neuron apoptosis in rats via regulating the Akt/mTOR pathway. Biosci Rep. 2019 Jan 22;39(1). doi: 10.1042/BSR-20181206_COR. Print 2019 Jan 31. PMID: 30670672; PMCID: PMC6347449.
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Identification of a Novel Mutation in the ABCA4 Gene in a Chinese Family with Retinitis Pigmentosa Using Exome Sequencing.

Bioscience reports

Huang X, Yuan L, Xu H, Zheng W, Cao Y, Yi J, Guo Y, Yang Z, Li Y, Deng H.
PMID: 29437900
Biosci Rep. 2018 Apr 27;38(2). doi: 10.1042/BSR20171300.

Retinitis pigmentosa (RP) is a group of hereditary, degenerative retinal disorders characterized by progressive retinal dysfunction, outer retina cell loss, and retinal tissue atrophy. It eventually leads to tunnel vision and legal, or total blindness. Here we aimed to...

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Huang X, Yuan L, Xu H, et al. Identification of a Novel Mutation in the ABCA4 Gene in a Chinese Family with Retinitis Pigmentosa Using Exome Sequencing. Biosci Rep. 2018;38(2)doi: 10.1042/BSR20171300.
Huang, X., Yuan, L., Xu, H., Zheng, W., Cao, Y., Yi, J., Guo, Y., Yang, Z., Li, Y., & Deng, H. (2018). Identification of a Novel Mutation in the ABCA4 Gene in a Chinese Family with Retinitis Pigmentosa Using Exome Sequencing. Bioscience reports, 38(2), . https://doi.org/10.1042/BSR20171300
Huang, Xiangjun, et al. "Identification of a Novel Mutation in the ABCA4 Gene in a Chinese Family with Retinitis Pigmentosa Using Exome Sequencing." Bioscience reports vol. 38,2 (2018). doi: https://doi.org/10.1042/BSR20171300
Huang X, Yuan L, Xu H, Zheng W, Cao Y, Yi J, Guo Y, Yang Z, Li Y, Deng H. Identification of a Novel Mutation in the ABCA4 Gene in a Chinese Family with Retinitis Pigmentosa Using Exome Sequencing. Biosci Rep. 2018 Apr 27;38(2). doi: 10.1042/BSR20171300. PMID: 29437900; PMCID: PMC5857910.
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Caspase-1 regulate AngII-induced cardiomyocyte hypertrophy via upregulation of IL-1β.

Bioscience reports

Bai Y, Sun X, Chu Q, Li A, Qin Y, Li Y, Yue E, Wang H, Li G, Zahra SM, Dong C, Jiang Y.
PMID: 29440460
Biosci Rep. 2018 Feb 12; doi: 10.1042/BSR20171438. Epub 2018 Feb 12.

Cardiac hypertrophy is a compensatory response to stress or stimuli, which results in arrhythmia and heart failure. Although multiple molecular mechanisms have been identified, cardiac hypertrophy is still difficult to treat. Pyroptosis is a caspase-1 dependent pro-inflammatory programmed cell...

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Bai Y, Sun X, Chu Q, et al. Caspase-1 regulate AngII-induced cardiomyocyte hypertrophy via upregulation of IL-1β. Biosci Rep. 2018;doi: 10.1042/BSR20171438.
Bai, Y., Sun, X., Chu, Q., Li, A., Qin, Y., Li, Y., Yue, E., Wang, H., Li, G., Zahra, S. M., Dong, C., & Jiang, Y. (2018). Caspase-1 regulate AngII-induced cardiomyocyte hypertrophy via upregulation of IL-1β. Bioscience reports, . https://doi.org/10.1042/BSR20171438
Bai, Yunlong, et al. "Caspase-1 regulate AngII-induced cardiomyocyte hypertrophy via upregulation of IL-1β." Bioscience reports vol. (2018). doi: https://doi.org/10.1042/BSR20171438
Bai Y, Sun X, Chu Q, Li A, Qin Y, Li Y, Yue E, Wang H, Li G, Zahra SM, Dong C, Jiang Y. Caspase-1 regulate AngII-induced cardiomyocyte hypertrophy via upregulation of IL-1β. Biosci Rep. 2018 Feb 12; doi: 10.1042/BSR20171438. Epub 2018 Feb 12. PMID: 29440460; PMCID: PMC5857903.
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Anarchy in the UPR: A Ca.

Bioscience reports

Schmitz-Peiffer C.
PMID: 29439143
Biosci Rep. 2018 Feb 08; doi: 10.1042/BSR20170966. Epub 2018 Feb 08.

Nonalcoholic fatty liver disease (NAFLD) is highly prevalent in Western countries, and is linked to the development of liver cancer and Type 2 diabetes (T2D). It is strongly associated with obesity, but the dysregulation of liver lipid storage is...

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Schmitz-Peiffer C. Anarchy in the UPR: A Ca. Biosci Rep. 2018;doi: 10.1042/BSR20170966.
Schmitz-Peiffer, C. (2018). Anarchy in the UPR: A Ca. Bioscience reports, . https://doi.org/10.1042/BSR20170966
Schmitz-Peiffer, Carsten. "Anarchy in the UPR: A Ca." Bioscience reports vol. (2018). doi: https://doi.org/10.1042/BSR20170966
Schmitz-Peiffer C. Anarchy in the UPR: A Ca. Biosci Rep. 2018 Feb 08; doi: 10.1042/BSR20170966. Epub 2018 Feb 08. PMID: 29439143; PMCID: PMC5857902.
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A modified, hypoallergenic variant of the  .

Bioscience reports

Pacheco-Soares T, de Oliveira Carvalho A, da Silva Araújo J, de Souza GDS, Machado OLT.
PMID: 29444820
Biosci Rep. 2018 Feb 14; doi: 10.1042/BSR20171245. Epub 2018 Feb 14.

Ric c1, an allergenic protein from

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Pacheco-Soares T, de Oliveira Carvalho A, da Silva Araújo J, et al. A modified, hypoallergenic variant of the  . Biosci Rep. 2018;doi: 10.1042/BSR20171245.
Pacheco-Soares, T., de Oliveira Carvalho, A., da Silva Araújo, J., de Souza, G. D. S., & Machado, O. L. T. (2018). A modified, hypoallergenic variant of the  . Bioscience reports, . https://doi.org/10.1042/BSR20171245
Pacheco-Soares, Thaís, et al. "A modified, hypoallergenic variant of the  ." Bioscience reports vol. (2018). doi: https://doi.org/10.1042/BSR20171245
Pacheco-Soares T, de Oliveira Carvalho A, da Silva Araújo J, de Souza GDS, Machado OLT. A modified, hypoallergenic variant of the  . Biosci Rep. 2018 Feb 14; doi: 10.1042/BSR20171245. Epub 2018 Feb 14. PMID: 29444820; PMCID: PMC5857906.
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Expression of Concern: MUC1 gene silencing inhibits proliferation, invasion and migration while promoting apoptosis of oral squamous cell carcinoma cells.

Bioscience reports

[No authors listed]
PMID: 32579205
Biosci Rep. 2020 Jun 26;40(6). doi: 10.1042/BSR-20182193_EOC.

No abstract available.

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Expression of Concern: MUC1 gene silencing inhibits proliferation, invasion and migration while promoting apoptosis of oral squamous cell carcinoma cells. Biosci Rep. 2020;40(6)doi: 10.1042/BSR-20182193_EOC.
(2020). Expression of Concern: MUC1 gene silencing inhibits proliferation, invasion and migration while promoting apoptosis of oral squamous cell carcinoma cells. Bioscience reports, 40(6), . https://doi.org/10.1042/BSR-20182193_EOC
"Expression of Concern: MUC1 gene silencing inhibits proliferation, invasion and migration while promoting apoptosis of oral squamous cell carcinoma cells." Bioscience reports vol. 40,6 (2020). doi: https://doi.org/10.1042/BSR-20182193_EOC
Expression of Concern: MUC1 gene silencing inhibits proliferation, invasion and migration while promoting apoptosis of oral squamous cell carcinoma cells. Biosci Rep. 2020 Jun 26;40(6). doi: 10.1042/BSR-20182193_EOC. PMID: 32579205; PMCID: PMC7316175.
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Retraction: miR-96-5p enhances cell proliferation and invasion via targeted regulation of ZDHHC5 in gastic cancer.

Bioscience reports

[No authors listed]
PMID: 32584392
Biosci Rep. 2020 Jun 26;40(6). doi: 10.1042/BSR-20191845_RET.

No abstract available.

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Retraction: miR-96-5p enhances cell proliferation and invasion via targeted regulation of ZDHHC5 in gastic cancer. Biosci Rep. 2020;40(6)doi: 10.1042/BSR-20191845_RET.
(2020). Retraction: miR-96-5p enhances cell proliferation and invasion via targeted regulation of ZDHHC5 in gastic cancer. Bioscience reports, 40(6), . https://doi.org/10.1042/BSR-20191845_RET
"Retraction: miR-96-5p enhances cell proliferation and invasion via targeted regulation of ZDHHC5 in gastic cancer." Bioscience reports vol. 40,6 (2020). doi: https://doi.org/10.1042/BSR-20191845_RET
Retraction: miR-96-5p enhances cell proliferation and invasion via targeted regulation of ZDHHC5 in gastic cancer. Biosci Rep. 2020 Jun 26;40(6). doi: 10.1042/BSR-20191845_RET. PMID: 32584392; PMCID: PMC7317580.
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Expression of Concern: Long non-coding RNA ROR1-AS1 incudes tumorigenesis of colorectal cancer by affecting Wnt/β-catenin signaling pathway.

Bioscience reports

[No authors listed]
PMID: 32633329
Biosci Rep. 2020 Jul 31;40(7). doi: 10.1042/BSR-20191453_EOC.

No abstract available.

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Expression of Concern: Long non-coding RNA ROR1-AS1 incudes tumorigenesis of colorectal cancer by affecting Wnt/β-catenin signaling pathway. Biosci Rep. 2020;40(7)doi: 10.1042/BSR-20191453_EOC.
(2020). Expression of Concern: Long non-coding RNA ROR1-AS1 incudes tumorigenesis of colorectal cancer by affecting Wnt/β-catenin signaling pathway. Bioscience reports, 40(7), . https://doi.org/10.1042/BSR-20191453_EOC
"Expression of Concern: Long non-coding RNA ROR1-AS1 incudes tumorigenesis of colorectal cancer by affecting Wnt/β-catenin signaling pathway." Bioscience reports vol. 40,7 (2020). doi: https://doi.org/10.1042/BSR-20191453_EOC
Expression of Concern: Long non-coding RNA ROR1-AS1 incudes tumorigenesis of colorectal cancer by affecting Wnt/β-catenin signaling pathway. Biosci Rep. 2020 Jul 31;40(7). doi: 10.1042/BSR-20191453_EOC. PMID: 32633329; PMCID: PMC7340864.
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Expression of Concern: Protective effect of autophagy on endoplasmic reticulum stress-induced apoptosis of alveolar epithelial cells in rat models of COPD.

Bioscience reports

[No authors listed]
PMID: 32716037
Biosci Rep. 2020 Jul 31;40(7). doi: 10.1042/BSR-20170803_EOC.

No abstract available.

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Expression of Concern: Protective effect of autophagy on endoplasmic reticulum stress-induced apoptosis of alveolar epithelial cells in rat models of COPD. Biosci Rep. 2020;40(7)doi: 10.1042/BSR-20170803_EOC.
(2020). Expression of Concern: Protective effect of autophagy on endoplasmic reticulum stress-induced apoptosis of alveolar epithelial cells in rat models of COPD. Bioscience reports, 40(7), . https://doi.org/10.1042/BSR-20170803_EOC
"Expression of Concern: Protective effect of autophagy on endoplasmic reticulum stress-induced apoptosis of alveolar epithelial cells in rat models of COPD." Bioscience reports vol. 40,7 (2020). doi: https://doi.org/10.1042/BSR-20170803_EOC
Expression of Concern: Protective effect of autophagy on endoplasmic reticulum stress-induced apoptosis of alveolar epithelial cells in rat models of COPD. Biosci Rep. 2020 Jul 31;40(7). doi: 10.1042/BSR-20170803_EOC. PMID: 32716037; PMCID: PMC7385586.
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Correction: The potential role of MGMT rs12917 polymorphism in cancer risk: an updated pooling analysis with 21010 cases and 34018 controls.

Bioscience reports

Sheng Z, Kang M, Wang H.
PMID: 30635450
Biosci Rep. 2019 Jan 11;39(1). doi: 10.1042/BSR-20180942_COR. Print 2019 Jan 31.

No abstract available.

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Sheng Z, Kang M, Wang H. Correction: The potential role of MGMT rs12917 polymorphism in cancer risk: an updated pooling analysis with 21010 cases and 34018 controls. Biosci Rep. 2019;39(1)doi: 10.1042/BSR-20180942_COR.
Sheng, Z., Kang, M., & Wang, H. (2019). Correction: The potential role of MGMT rs12917 polymorphism in cancer risk: an updated pooling analysis with 21010 cases and 34018 controls. Bioscience reports, 39(1), . https://doi.org/10.1042/BSR-20180942_COR
Sheng, Zhiguo, et al. "Correction: The potential role of MGMT rs12917 polymorphism in cancer risk: an updated pooling analysis with 21010 cases and 34018 controls." Bioscience reports vol. 39,1 (2019). doi: https://doi.org/10.1042/BSR-20180942_COR
Sheng Z, Kang M, Wang H. Correction: The potential role of MGMT rs12917 polymorphism in cancer risk: an updated pooling analysis with 21010 cases and 34018 controls. Biosci Rep. 2019 Jan 11;39(1). doi: 10.1042/BSR-20180942_COR. Print 2019 Jan 31. PMID: 30635450; PMCID: PMC6347448.
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Showing 1 to 12 of 820 entries